Genetic polymorphism of ACE and the angiotensin II type1 receptor genes in children with chronic kidney disease
2011

Genetic Factors in Kidney Disease in Children

Sample size: 76 publication 10 minutes Evidence: moderate

Author Information

Author(s): Elshamaa Manal F, Sabry Samar M, Bazaraa Hafez M, Koura Hala M, Elghoroury Eman A, Kantoush Nagwa A, Thabet Eman H, Abd-El Haleem Dalia A

Primary Institution: National Research Centre, Cairo, Egypt

Hypothesis

The study investigates the association between polymorphisms of the ACE and AT1R genes and the occurrence of renal disease in children.

Conclusion

The ACE DD and AT1R A/C genotypes are associated with hypertension and renal damage in children with end-stage renal disease.

Supporting Evidence

  • ACE DD genotype was significantly more frequent in MHD patients compared to controls.
  • 89% of DD genotype patients were hypertensive compared to 61% of non-DD genotype patients.
  • The C allele of AT1R was independently associated with hypertension.
  • ACE activity was higher in patients with the DD genotype.
  • Patients with the C allele had higher serum ACE activity.

Takeaway

This study looked at how certain genes might make kids with kidney disease more likely to have high blood pressure and other problems.

Methodology

The study included 76 pediatric patients with advanced CKD and 70 healthy controls, measuring serum ACE activity and genotyping for ACE and AT1R polymorphisms.

Potential Biases

Potential bias due to the small sample size and selection from a single center.

Limitations

The study had a small sample size and was conducted at a single center, which may limit the generalizability of the findings.

Participant Demographics

Pediatric patients with advanced chronic kidney disease, ages not specified.

Statistical Information

P-Value

0.0001

Confidence Interval

95% CI (0.07-0.24)

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1476-9255-8-20

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication