Protein interactions in human genetic diseases
2008

Protein Interactions in Human Genetic Diseases

Sample size: 1428 publication Evidence: moderate

Author Information

Author(s): Schuster-Böckler Benjamin, Bateman Alex

Primary Institution: Wellcome Trust Sanger Institute

Hypothesis

Can a method combining protein structure information with interaction data predict mutations related to interaction defects?

Conclusion

The study identified 1,428 mutations likely related to protein interaction defects, suggesting that about 4% of mutations could affect protein interactions.

Supporting Evidence

  • The method predicted 1,428 mutations related to interaction defects.
  • Approximately 4% of all mutations could be linked to protein interactions.
  • The study provides a curated list of 119 interaction-related mutations from the literature.

Takeaway

The researchers found that some changes in our genes can mess up how proteins interact, which might cause diseases.

Methodology

A novel method was developed to identify residues involved in protein interactions by combining structural and experimental data.

Potential Biases

Potential bias in the selection of mutations from databases and literature.

Limitations

The study primarily focused on single-residue mutations and may not capture all interaction-related mutations.

Statistical Information

P-Value

< 0.014

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/gb-2008-9-1-r9

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