Sinomenine inhibits microglial activation by Aβ and confers neuroprotection
2011

Sinomenine's Role in Protecting Brain Cells from Alzheimer's Damage

Sample size: 7 publication Evidence: moderate

Author Information

Author(s): Shilpa Mishra Shukla, Shiv K Sharma

Primary Institution: National Brain Research Centre, Manesar, Haryana, India

Hypothesis

Does sinomenine prevent microglial activation and neurotoxicity induced by amyloid-β?

Conclusion

Sinomenine prevents microglial activation and protects hippocampal cells from indirect neurotoxicity caused by amyloid-β.

Supporting Evidence

  • Sinomenine reduces levels of reactive oxygen species and nitric oxide in microglial cells.
  • Sinomenine protects hippocampal HT22 cells from indirect toxicity mediated by Aβ-treated microglial cells.
  • Sinomenine inhibits the release of inflammatory molecules induced by oligomeric Aβ.

Takeaway

Sinomenine is a natural compound that helps protect brain cells from damage caused by a harmful protein linked to Alzheimer's disease.

Methodology

The study involved treating BV2 microglial cells and hippocampal HT22 cells with sinomenine and oligomeric amyloid-β, followed by various assays to measure reactive oxygen species, nitric oxide, and cell viability.

Limitations

Sinomenine did not protect against direct toxicity from amyloid-β in hippocampal cells.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1742-2094-8-117

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