Retinoblastoma Makes Its Mark on Imprinting in Plants
2008

Retinoblastoma and Its Role in Plant Imprinting

publication Evidence: moderate

Author Information

Author(s): Liliana M. Costa, José F. Gutierrez-Marcos

Primary Institution: University of Oxford

Hypothesis

Do plant imprints also passively lose methylation during the cell cycle through a similar process as in mammals?

Conclusion

The Retinoblastoma pathway is an important regulatory component in the establishment of genomic imprinting in plants.

Supporting Evidence

  • The RB-MSI1 complex may interact with E2F transcription factors and HDACs to repress the expression of the DNA methyltransferase MET1.
  • Mutations in imprinted PcG genes hint that the RB-MSI1 complex might be controlled by components of the PcG pathway.
  • Direct binding of RB and MSI1 to the MET1 promoter suggests regulation of DNA methyltransferase activity during the cell cycle.

Takeaway

This study shows that a protein involved in cancer regulation in animals also helps control how plants express certain genes during reproduction.

Methodology

The authors explored the role of the RB-MSI1 pathway in regulating imprinting through repression of DNA methylation during female gametogenesis.

Limitations

The study does not fully resolve how the RB-MSI1 complex associates in vivo during female gametogenesis.

Digital Object Identifier (DOI)

10.1371/journal.pbio.0060212

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