Tenascin-C Enhances Pancreatic Cancer Cell Growth and Motility and Affects Cell Adhesion through Activation of the Integrin Pathway
2011

Tenascin-C and Its Role in Pancreatic Cancer

publication Evidence: moderate

Author Information

Author(s): Paron Igor, Berchtold Sonja, Vörös Julia, Shamarla Madhavi, Erkan Mert, Höfler Heinz, Esposito Irene

Primary Institution: Institute of Pathology, Helmholtz Zentrum München, German Research Center for Environmental Health, Neuherberg, Germany

Hypothesis

The study aims to characterize the effects of Tenascin-C (TNC) on the biological properties of pancreatic cancer cells.

Conclusion

TNC affects proliferation, migration, and adhesion of poorly differentiated pancreatic cancer cell lines and might therefore play a role in PDAC spreading and metastasis in vivo.

Supporting Evidence

  • Endogenous TNC promoted pancreatic cancer cell growth and migration.
  • A TNC-rich matrix enhanced migration and adhesion to uncoated surfaces.
  • TNC decreased adhesion to fibronectin in pancreatic cancer cells.

Takeaway

Tenascin-C helps pancreatic cancer cells grow and move around, which might make the cancer spread more easily.

Methodology

The study used proliferation, migration, and adhesion assays on pancreatic cancer cell lines treated with TNC or grown on a TNC-rich matrix.

Limitations

The effects of TNC on cell viability and proliferation were heterogeneous across different cell lines.

Statistical Information

P-Value

p<0.001

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0021684

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication