Disruption of the thrombospondin-2 gene alters the lamellar morphology but does not permit vascularization of the adult mouse lumbar disc
2008

Impact of Thrombospondin-2 Gene Disruption on Mouse Lumbar Disc Structure

Sample size: 8 publication 10 minutes Evidence: moderate

Author Information

Author(s): Helen E Gruber, Paul Bornstein, Helene E Sage, Jane A Ingram, Natalia Zinchenko, H James Norton, Edward N Hanley Jr

Primary Institution: Carolinas Medical Center

Hypothesis

Does the disruption of the thrombospondin-2 gene enhance vascularity in the adult annulus of mice?

Conclusion

The study confirms that thrombospondin-2 plays a role in the morphology of the disc but does not allow for vascular ingrowth into the disc.

Supporting Evidence

  • TSP-2 was present in some annulus cells of both human and mouse discs.
  • TSP-2-null mice showed a more irregular collagen structure in their discs compared to wild-type mice.
  • Vascular beds in TSP-2-null mice were significantly larger than in wild-type mice.
  • No vascular ingrowth into the discs of TSP-2-null mice was observed.

Takeaway

The researchers looked at mice without a specific gene and found that while there were more blood vessels around the disc, they didn't grow into the disc itself.

Methodology

The study used immunohistochemistry and quantitative histology to examine the expression and location of TSP-2 in human and mouse discs.

Limitations

The study did not measure nutrient diffusion rates in the discs.

Participant Demographics

Human disc tissues were derived from individuals with herniated discs and degenerative disc disease, and mouse subjects were 5-month-old wild-type and TSP-2-null mice.

Statistical Information

P-Value

0.0002

Statistical Significance

p=0.0002

Digital Object Identifier (DOI)

10.1186/ar2483

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