PPARs and Bone Metabolism
2006

PPARs and Bone Metabolism

Editorial

Author Information

Author(s): Beata Lecka-Czernik

Primary Institution: University of Arkansas for Medical Sciences

Hypothesis

PPAR-γ acts as a regulator of chondrocyte development and cartilage homeostasis.

Conclusion

PPAR-γ plays a key role in maintaining bone health during aging and disease.

Supporting Evidence

  • PPAR-γ is involved in mesenchymal stem cell lineage allocation.
  • There is possible cross-talk between PPAR-γ and relevant nuclear receptors.
  • PPAR-γ gene polymorphisms are examined in relation to bone mineral density in humans.
  • PPAR-γ is linked to bone loss due to skeletal disuse.
  • Human bone is vulnerable to antidiabetic therapies with PPAR-γ agonists.
  • The antiosteoblastic activity of PPAR-γ can be separated from its other activities using selective modulators.

Takeaway

PPARs are important for keeping our bones healthy, especially as we get older.

Limitations

Not all aspects of PPARs involvement in bone maintenance are covered.

Digital Object Identifier (DOI)

10.1155/PPAR/2006/18089

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