Comparative Expression Profile of miRNA and mRNA in Primary Peripheral Blood Mononuclear Cells Infected with Human Immunodeficiency Virus (HIV-1)
2011

Comparative Expression Profile of miRNA and mRNA in HIV-1 Infected Blood Cells

Sample size: 16 publication 10 minutes Evidence: moderate

Author Information

Author(s): Ankit Gupta, Pruthvi Nagilla, Le Hai-Son, Coulton Bunney, Courtney Zych, Anbupalam Thalamuthu, Ziv Bar-Joseph, Sinnakaruppan Mathavan, Velpandi Ayyavoo

Primary Institution: University of Pittsburgh

Hypothesis

HIV-1 infection may regulate an identical subset of miRNA in cells derived from genetically diverse individuals, impacting mRNA in genes associated with distinct cellular pathways.

Conclusion

HIV-1 infection alters the regulation of 21 miRNAs and 444 mRNAs, indicating that the miRNA profile could serve as an early indicator of host cellular dysfunction.

Supporting Evidence

  • HIV-1 infection led to altered regulation of 21 miRNAs and 444 mRNAs.
  • The differentially regulated miRNA and mRNA were associated with pathways involved in cell cycle, apoptosis, T-cell signaling, and immune activation.
  • Results indicate that the miRNA profile could be an early indicator of host cellular dysfunction induced by HIV-1.

Takeaway

When people get infected with HIV, their blood cells change how they use tiny molecules called miRNAs and mRNAs, which help control how genes work.

Methodology

The study used a comparative global miRNA and mRNA microarray profiling in PBMCs from multiple donors infected with HIV-1.

Potential Biases

Potential biases may arise from using PBMCs from healthy donors, which may not fully represent the infected population.

Limitations

The study may not account for variability in host genetics and viral load among different individuals.

Participant Demographics

PBMCs were derived from multiple healthy donors who were seronegative for HIV-1.

Statistical Information

P-Value

<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0022730

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