FGFR1 amplification in breast carcinomas: a chromogenic in situ hybridisation analysis
2007

FGFR1 Amplification in Breast Cancer

Sample size: 880 publication 10 minutes Evidence: moderate

Author Information

Author(s): Elbauomy Elsheikh, Andrew R Green, Maryou BK Lambros, Nicholas C Turner, Matthew J Grainge, Des Powe, Ian O Ellis, Jorge S Reis-Filho

Primary Institution: Nottingham University Hospitals Trust and University of Nottingham

Hypothesis

Does FGFR1 amplification correlate with clinical outcomes in breast cancer?

Conclusion

FGFR1 amplification is found in 8.7% of breast cancers and is an independent predictor of overall survival.

Supporting Evidence

  • FGFR1 amplification was observed in 8.7% of the tumors.
  • FGFR1 amplification was significantly more prevalent in patients over 50 years of age.
  • FGFR1 amplification was the strongest independent predictor of poor outcome in estrogen-receptor-positive tumors.

Takeaway

Some breast cancers have a change in a gene called FGFR1, which can make them harder to treat, especially in older patients.

Methodology

FGFR1 amplification was analyzed in tissue microarrays of 880 breast tumors using chromogenic in situ hybridization.

Limitations

The study did not correlate FGFR1 amplification with protein expression due to antibody optimization issues.

Participant Demographics

Cohort included unselected breast tumors from patients presenting between 1986 and 1998.

Statistical Information

P-Value

p<0.05

Confidence Interval

95%

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/bcr1665

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