Polymorphisms in Nucleotide Excision Repair Genes, Arsenic Exposure, and Non-Melanoma Skin Cancer in New Hampshire
2007

Arsenic Exposure and Skin Cancer Risk in New Hampshire

Sample size: 2280 publication 10 minutes Evidence: moderate

Author Information

Author(s): Katie M. Applebaum, Margaret R. Karagas, David J. Hunter, Paul J. Catalano, Steven H. Byler, Steve Morris, Heather H. Nelson

Primary Institution: Harvard School of Public Health

Hypothesis

Do polymorphisms in nucleotide excision repair genes modify the association between arsenic exposure and non-melanoma skin cancer?

Conclusion

The study suggests that certain genetic variants may reduce the risk of non-melanoma skin cancer in relation to arsenic exposure.

Supporting Evidence

  • High arsenic exposure was linked to increased basal cell carcinoma risk among those with specific genetic variants.
  • The study included 880 cases of basal cell carcinoma and 666 cases of squamous cell carcinoma.
  • Participants were matched on age and sex to ensure a representative control group.

Takeaway

This study looked at how some people's genes might change their risk of getting skin cancer from arsenic in drinking water.

Methodology

The study involved identifying skin cancer cases through dermatologists and pathology labs, with controls matched by age and sex, and measuring arsenic exposure through toenail clippings.

Potential Biases

Potential reporting biases were minimized by blinding interviewers and participants to case-control status.

Limitations

The study is limited by its reliance on toenail arsenic as a single point measure of exposure.

Participant Demographics

Participants were primarily Caucasian, aged 25 to 74, with a majority being male.

Statistical Information

P-Value

0.07

Confidence Interval

95% CI, 0.9–3.7

Statistical Significance

p<0.07

Digital Object Identifier (DOI)

10.1289/ehp.10096

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