Cigarette smoke extract upregulates heme oxygenase-1 via PKC/NADPH oxidase/ROS/PDGFR/PI3K/Akt pathway in mouse brain endothelial cells
2011

Cigarette Smoke Extract and Heme Oxygenase-1 in Brain Cells

publication Evidence: moderate

Author Information

Author(s): Shih Ruey-Horng, Cheng Shin-Ei, Hsiao Li-Der, Kou Yu Ru, Yang Chuen-Mao

Primary Institution: Chang Gung University

Hypothesis

The study investigates the mechanisms by which cigarette smoke extract modulates heme oxygenase-1 protein expression in cerebral endothelial cells.

Conclusion

Cigarette smoke extract induces heme oxygenase-1 expression through a specific signaling pathway involving PKCδ and NADPH oxidase in brain endothelial cells.

Supporting Evidence

  • Cigarette smoke extract significantly induced HO-1 protein expression in brain endothelial cells.
  • The induction of HO-1 was mediated through a PKCδ/NADPH oxidase/ROS signaling pathway.
  • Inhibitors of PKCδ and NADPH oxidase reduced the expression of HO-1 in response to cigarette smoke extract.

Takeaway

Cigarette smoke can make brain cells produce a protein that can be harmful, and this happens through a specific process in the cells.

Methodology

The study used cultured mouse brain endothelial cells to assess the effects of cigarette smoke extract on heme oxygenase-1 expression and the underlying molecular mechanisms.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1742-2094-8-104

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