High-fat diet mouse model receiving L-glucose supplementations propagates liver injury
2024

Effects of L-Glucose on Liver Injury in Mice

Sample size: 60 publication 10 minutes Evidence: moderate

Author Information

Author(s): Amer Johnny, Amleh Athar, Salhab Ahmad, Kolodny Yuval, Yochelis Shira, Saffouri Baker, Paltiel Yossi, Safadi Rifaat

Primary Institution: Liver Institute, Hadassah-Hebrew University Hospital, Jerusalem, Israel

Hypothesis

How do manufactured sugars like L-glucose affect liver injury compared to natural sugars like D-glucose in high-fat diet mouse models?

Conclusion

L-glucose supplementation in high-fat diet mice leads to less liver fibrosis and increased lipid clearance compared to D-glucose.

Supporting Evidence

  • D- and L-Glu supplementations increased liver inflammation and fibrosis in high-fat diet-fed mice.
  • L-Glu supplementation led to a significant reduction in liver weights in high-fat diet-fed mice.
  • Both D- and L-Glu caused increased serum malondialdehyde levels, indicating lipid peroxidation.
  • HOMA-IR scores increased in high-fat diet-fed mice with glucose supplementations.
  • L-Glu supplementation resulted in less hepatic lipid uptake compared to D-Glu.

Takeaway

This study shows that giving mice a type of artificial sugar called L-glucose can help their livers get rid of fat better than a natural sugar called D-glucose.

Methodology

C57BL/6 mice were fed either a regular diet or a high-fat diet for 16 weeks, with L-glucose or D-glucose added to their drinking water from weeks 8 to 16.

Potential Biases

Potential bias in the selection of glucose types and their effects on liver injury.

Limitations

The study is limited to a mouse model and may not fully represent human metabolic responses.

Participant Demographics

C57BL/6 male mice, aged six weeks at the start of the study.

Statistical Information

P-Value

p<0.002

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.3389/fnut.2024.1469952

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication