Dermal-Type Macrophages Expressing CD209/DC-SIGN Show Inherent Resistance to Dengue Virus Growth
2008

Dermal Macrophages Resist Dengue Virus Growth

Sample size: 6 publication 10 minutes Evidence: moderate

Author Information

Author(s): Kwan Wing-Hong, Navarro-Sanchez Erika, Dumortier Hélène, Decossas Marion, Vachon Hortense, dos Santos Flavia Barreto, Fridman Hervé W., Rey Félix A., Harris Eva, Despres Philippe, Mueller Christopher G.

Primary Institution: CNRS, Laboratory of Therapeutic Immunology and Chemistry, IBMC, Université Louis Pasteur, Strasbourg, France

Hypothesis

Are dermal macrophages resistant to dengue virus infection?

Conclusion

Dermal macrophages can internalize dengue virus but do not produce progeny virus due to the accumulation of the virus in poorly-acidified phagosomes.

Supporting Evidence

  • Dermal macrophages express CD209 and can bind dengue virus envelope protein.
  • Monocyte-derived dermal macrophages were resistant to dengue virus replication.
  • Dengue virus was internalized but not produced in dermal macrophages.
  • Accumulation of the virus in poorly-acidified phagosomes prevented infection.

Takeaway

The study found that special immune cells in the skin called dermal macrophages can catch the dengue virus but don't let it grow, which helps protect the body from getting sick.

Methodology

The researchers generated dermal-type macrophages from monocytes and tested their ability to bind and internalize dengue virus.

Potential Biases

Potential bias in the selection of healthy donors for skin samples.

Limitations

The study did not explore the long-term effects of dengue virus on dermal macrophages or the potential for other pathogens to exploit these cells.

Participant Demographics

Healthy adult human donors undergoing plastic surgery.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pntd.0000311

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