Central Exercise Action Increases the AMPK and mTOR Response to Leptin Exercise and Leptin Action
2008

Exercise Enhances Leptin's Effects on Appetite Control

Sample size: 12 publication 10 minutes Evidence: moderate

Author Information

Author(s): Ropelle Eduardo R., Fernandes Maria Fernanda A., Flores Marcelo B. S., Ueno Mirian, Rocco Silvana Marin, Cintra Rodrigo, Velloso Dennys E., Franchini Lício A., Saad Kleber G., Carvalheira Mario J. A., José B. C.

Primary Institution: State University of Campinas (UNICAMP), Campinas, São Paulo, Brazil

Hypothesis

Does exercise improve the AMPK and mTOR response to leptin in the hypothalamus in an IL-6-dependent manner?

Conclusion

Exercise improves the AMPK and mTOR responses to leptin administration, contributing to appetite-suppressive actions.

Supporting Evidence

  • IL-6 infusion reduced food intake in rats.
  • Exercise increased the sensitivity to leptin's effects.
  • AMPK and mTOR pathways were more responsive after exercise.
  • Blocking IL-6 reduced the effectiveness of leptin.
  • Exercise did not change food intake directly but enhanced leptin's effects.

Takeaway

When rats exercise, their bodies respond better to a hormone called leptin that helps control hunger, which might help prevent obesity.

Methodology

Rats performed two 3-hour exercise bouts, and their food intake and signaling pathways were analyzed after IL-6 infusion.

Limitations

The study does not establish the mechanism by which long-term exercise decreases leptin levels while increasing the response to leptin.

Participant Demographics

Male Wistar rats, 8 weeks old, weighing 250-300 g.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0003856

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication