A genome-wide association study for blood lipid phenotypes in the Framingham Heart Study
2007

Genetic Study of Blood Lipids in the Framingham Heart Study

Sample size: 1087 publication Evidence: moderate

Author Information

Author(s): Kathiresan Sekar, Manning Alisa K, Demissie Serkalem, D'Agostino Ralph B, Surti Aarti, Guiducci Candace, Gianniny Lauren, Burtt Nöel P, Melander Olle, Orho-Melander Marju, Arnett Donna K, Peloso Gina M, Ordovas Jose M, Cupples L Adrienne

Primary Institution: National Heart Lung and Blood Institute's Framingham Heart Study

Hypothesis

Common genetic variants explain a proportion of the inter-individual variability in LDL-C, HDL-C, and TG.

Conclusion

The study did not identify any new genetic loci associated with blood lipid variability despite initial promising associations.

Supporting Evidence

  • Long-term averages of LDL-C, HDL-C, and TG were highly heritable.
  • Two SNPs had p < 10-5 in GEE results for LDL-C.
  • Validation in additional samples did not identify any new loci underlying variability in blood lipids.

Takeaway

Scientists looked at people's genes to see how they affect cholesterol and fat levels in the blood, but they didn't find any new important genes that explain these differences.

Methodology

Genome-wide association study using Affymetrix 100K GeneChip to analyze blood lipid traits in Framingham Heart Study participants.

Potential Biases

Potential for false positives due to the large number of tests conducted.

Limitations

Limited statistical power to detect common SNPs explaining less than 1% of trait variance and confinement to a single ancestral group.

Participant Demographics

Mean age 47 years, 52% women.

Statistical Information

P-Value

p<10-5

Statistical Significance

p<0.0001

Digital Object Identifier (DOI)

10.1186/1471-2350-8-S1-S17

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