Ambient particulate air pollution induces oxidative stress and alterations of mitochondria and gene expression in brown and white adipose tissues
2011

Air Pollution Affects Fat Tissue and Metabolism in Mice

Sample size: 8 publication 10 minutes Evidence: moderate

Author Information

Author(s): Xu Zhaobin, Xu Xiaohua, Zhong Mianhua, Hotchkiss Ian P, Lewandowski Ryan P, Wagner James G, Bramble Lori A, Yang Yifeng, Wang Aixia, Harkema Jack R, Lippmann Morton, Rajagopalan Sanjay, Chen Lung-Chi, Sun Qinghua

Primary Institution: The Ohio State University

Hypothesis

PM2.5 exposure may have differential effects on brown and white adipose tissues.

Conclusion

Exposure to PM2.5 triggers oxidative stress in brown adipose tissue and alters mitochondrial gene expression.

Supporting Evidence

  • PM2.5 exposure increased reactive oxygen species production in brown adipose tissue.
  • Mitochondrial number was significantly reduced in both white and brown adipose tissues.
  • UCP1 expression was significantly decreased in brown adipose tissue after PM2.5 exposure.
  • Gene expression of brown adipocyte-specific genes was down-regulated in response to PM2.5.

Takeaway

Breathing in polluted air can hurt the fat in our bodies, making it less healthy and affecting how we use energy.

Methodology

Male ApoE knockout mice inhaled PM2.5 or filtered air for 6 hours a day, 5 days a week, for 2 months, and various analyses were performed on adipose tissues.

Limitations

The study was limited by sample size and tissue availability.

Participant Demographics

Male ApoE knockout mice, 4 weeks old.

Statistical Information

P-Value

<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1743-8977-8-20

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