Auto-Ubiquitination-Induced Degradation of MALT1-API2 Prevents BCL10 Destabilization in t(11;18)(q21;q21)-Positive MALT Lymphoma
2009

MALT1-API2 Fusion in MALT Lymphoma

Sample size: 14 publication 10 minutes Evidence: moderate

Author Information

Author(s): Noels Heidi, Somers Riet, Liu Hongxiang, Ye Hongtao, Du Ming-Qing, De Wolf-Peeters Christiane, Marynen Peter, Baens Mathijs

Primary Institution: Human Genome Laboratory, Department for Molecular and Developmental Genetics, VIB, Leuven, Belgium

Hypothesis

Does the MALT1-API2 fusion protein affect BCL10 levels and NF-κB activation in MALT lymphoma?

Conclusion

MALT1-API2 is unstable and does not significantly affect BCL10 levels or NF-κB activation in MALT lymphoma.

Supporting Evidence

  • MALT1-API2 transcripts were found in 8 out of 14 MALT lymphoma cases.
  • The RING domain of MALT1-API2 allows it to function as an E3 ubiquitin ligase.
  • MALT1-API2 does not significantly affect BCL10 protein levels in vivo.
  • Expression of MALT1-API2 reduces NF-κB activation by API2-MALT1.

Takeaway

This study looks at a protein involved in a type of cancer and finds that it doesn't really change how much of another important protein is present.

Methodology

RT-PCR and sequencing were used to analyze MALT1-API2 expression in MALT lymphoma samples.

Limitations

The study could not confirm the effect of MALT1-API2 on BCL10 levels in all cases due to insufficient material.

Participant Demographics

MALT lymphoma cases with t(11;18)(q21;q21) translocation.

Digital Object Identifier (DOI)

10.1371/journal.pone.0004822

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