Non-Lytic, Actin-Based Exit of Intracellular Parasites from C. elegans Intestinal Cells
2011

Non-Lytic Exit of Parasites from Intestinal Cells

Sample size: 164 publication 10 minutes Evidence: high

Author Information

Author(s): Kathleen A. Estes, Suzannah C. Szumowski, Emily R. Troemel

Primary Institution: University of California, San Diego

Hypothesis

How does the microsporidian parasite N. parisii exit C. elegans intestinal cells without causing cell lysis?

Conclusion

N. parisii uses a novel two-phase, non-lytic exit strategy that involves actin rearrangement and terminal web restructuring in host intestinal cells.

Supporting Evidence

  • N. parisii infection causes actin to relocate within intestinal cells.
  • Gaps in the terminal web appear before spore exit.
  • All contagious animals exhibit gaps in the terminal web.
  • Reducing actin levels impairs spore exit.
  • Spore exit does not cause lysis of intestinal cells.

Takeaway

The parasite N. parisii can leave the intestinal cells of C. elegans without breaking them, which helps it spread to other worms.

Methodology

The study involved infecting C. elegans with N. parisii spores and analyzing the actin and terminal web structures during infection.

Limitations

The study primarily focuses on a single host-parasite interaction and may not generalize to other systems.

Participant Demographics

C. elegans nematodes were used as the model organism.

Statistical Information

P-Value

p<0.0005

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.ppat.1002227

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication