Amitriptyline-Mediated Cognitive Enhancement in Aged 3×Tg Alzheimer's Disease Mice Is Associated with Neurogenesis and Neurotrophic Activity
2011

Amitriptyline Improves Memory in Alzheimer's Mice

Sample size: 30 publication 10 minutes Evidence: high

Author Information

Author(s): Chadwick Wayne, Mitchell Nick, Caroll Jenna, Zhou Yu, Park Sung-Soo, Wang Liyun, Becker Kevin G., Zhang Yongqing, Lehrmann Elin, Wood William H. III, Martin Bronwen, Maudsley Stuart

Primary Institution: National Institute on Aging, National Institutes of Health, Baltimore, Maryland, United States of America

Hypothesis

Can amitriptyline enhance cognitive function in aged 3×Tg Alzheimer's disease mice?

Conclusion

Amitriptyline treatment significantly improved memory and cognitive function in aged 3×TgAD mice.

Supporting Evidence

  • Amitriptyline treatment improved both long- and short-term memory retention in mice.
  • Neurogenesis was significantly increased in the dentate gyrus of treated mice.
  • Amitriptyline reduced levels of toxic amyloid beta dimers in the brain.
  • Significant increases in neurosynaptic marker proteins were observed after treatment.
  • Behavioral tests showed that treated mice remembered the location of a hidden platform better than controls.

Takeaway

Amitriptyline, a common antidepressant, helped older mice with Alzheimer's remember things better.

Methodology

Aged 3×TgAD mice were treated with amitriptyline for 4 months, followed by cognitive behavior testing and analysis of brain tissue.

Potential Biases

Potential bias in behavioral testing due to the subjective nature of some assessments.

Limitations

The study was conducted on a specific mouse model, which may not fully represent human Alzheimer's disease.

Participant Demographics

Aged male 3×TgAD mice, 14 months old.

Statistical Information

P-Value

p=0.00098

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0021660

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