Genes of the Unfolded Protein Response Pathway Harbor Risk Alleles for Primary Open Angle Glaucoma
2011

Genes Linked to Primary Open Angle Glaucoma

Sample size: 678 publication 10 minutes Evidence: moderate

Author Information

Author(s): Carbone Mary Anna, Chen Yuhong, Hughes Guy A., Weinreb Robert N., Zabriskie Norman A., Zhang Kang, Anholt Robert R. H.

Primary Institution: North Carolina State University

Hypothesis

Can a comparative genomics approach identify risk alleles for primary open angle glaucoma (POAG) in human populations?

Conclusion

The study identified two genes, BIRC6 and PDIA5, that harbor risk alleles for POAG, implicating the unfolded protein response in its pathogenesis.

Supporting Evidence

  • Two genes, BIRC6 and PDIA5, were found to have significant associations with POAG.
  • The study used a Drosophila model to identify candidate genes related to the unfolded protein response.
  • Significant SNPs were replicated in two independent populations.
  • Findings suggest that the unfolded protein response may play a role in the pathogenesis of POAG.

Takeaway

Scientists found two genes that might make people more likely to get a type of eye disease called glaucoma, which can cause blindness.

Methodology

The study used a comparative genomics approach with a transgenic Drosophila model and case-control studies in two Caucasian populations.

Potential Biases

Potential population-specific variants may not be captured due to the focus on Caucasian populations.

Limitations

The study's sample size was limited compared to other genome-wide association studies, which may affect the generalizability of the findings.

Participant Demographics

Participants were of Caucasian ethnicity, with mean ages of 69 for controls and 76 for cases in San Diego, and 68 for controls and 74 for cases in Salt Lake City.

Statistical Information

P-Value

5.84×10–9

Confidence Interval

95% CI 1.6–3.5

Statistical Significance

p<0.0008

Digital Object Identifier (DOI)

10.1371/journal.pone.0020649

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication