Functional Hair Cell Mechanotransducer Channels Are Required for Aminoglycoside Ototoxicity
2011

How Aminoglycosides Enter Hair Cells and Cause Hearing Loss

publication 10 minutes Evidence: high

Author Information

Author(s): Alharazneh Abdelrahman, Luk Lauren, Huth Markus, Monfared Ashkan, Steyger Peter S., Cheng Alan G., Ricci Anthony J.

Primary Institution: Stanford University

Hypothesis

The study investigates the mechanisms of aminoglycoside transport in mammalian hair cells, focusing on mechanotransducer channels and endocytosis.

Conclusion

The study concludes that mechanotransducer channels are primarily responsible for aminoglycoside entry into hair cells, leading to cell death and hearing loss.

Supporting Evidence

  • Aminoglycosides cause hair cell death by entering through mechanotransducer channels.
  • Hypocalcemia increases the entry of aminoglycosides into hair cells.
  • Blockers of mechanotransducer channels significantly reduce aminoglycoside uptake.
  • Curare and quinine protect hair cells from aminoglycoside toxicity.

Takeaway

Aminoglycoside antibiotics can get into hearing cells through special channels, which can cause those cells to die and lead to hearing loss.

Methodology

The study used rat cochlear explants cultured in gentamicin-containing media and employed two-photon imaging to observe drug uptake.

Limitations

The study cannot rule out other potential transport mechanisms for aminoglycosides under different pathological conditions.

Participant Demographics

Rat cochlear explants from postnatal 4-day-old pups were used in the experiments.

Statistical Information

P-Value

p<0.001

Statistical Significance

p<0.001

Digital Object Identifier (DOI)

10.1371/journal.pone.0022347

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication