Spleen Vagal Denervation Inhibits the Production of Antibodies to Circulating Antigens Vagus Induced IgM Production
2008

How the Brain Influences Antibody Production

Sample size: 12 publication 10 minutes Evidence: high

Author Information

Author(s): Ruud M. Buijs, Jan van der Vliet, Mari-Laure Garidou, Inge Huitinga, Carolina Escobar

Primary Institution: Instituto de Investigaciones Biomédicas, Universidad Nacional Autónoma de México

Hypothesis

Can the brain stimulate the production of antibodies through its autonomic output?

Conclusion

The study shows that the brain can stimulate antibody production by its autonomic output, particularly through parasympathetic innervation of the spleen.

Supporting Evidence

  • The spleen receives both sympathetic and parasympathetic input from the brain.
  • Injection of LPS with TNP-OVA activates the brain and induces specific IgM production.
  • Parasympathetic denervation of the spleen eliminates the antibody response to TNP-OVA.

Takeaway

The brain helps the body fight infections by telling the spleen to make antibodies, which are like little soldiers that protect us from getting sick.

Methodology

The study used anatomical and physiological techniques, including retrograde tracing and antibody measurement after specific denervation of the spleen.

Potential Biases

Potential bias in the interpretation of the role of the autonomic nervous system in immune response due to the focus on specific pathways.

Limitations

The study primarily focused on the parasympathetic nervous system and did not explore other potential mechanisms of immune response.

Participant Demographics

Rats were used as the animal model for the experiments.

Statistical Information

P-Value

p<0.01

Statistical Significance

p<0.01

Digital Object Identifier (DOI)

10.1371/journal.pone.0003152

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication