Astrocyte-Derived Tissue Transglutaminase Interacts with Fibronectin: A Role in Astrocyte Adhesion and Migration?
2011

Astrocyte-Derived Tissue Transglutaminase and Its Role in Astrocyte Adhesion and Migration

Sample size: 6 publication 10 minutes Evidence: moderate

Author Information

Author(s): van Strien Miriam E., Brevé John J. P., Fratantoni Silvina, Schreurs Marco W. J., Bol John G. J. M., Jongenelen Cornelis A. M., Drukarch Benjamin, van Dam Anne-Marie

Primary Institution: VU University Medical Center, Neuroscience Campus Amsterdam, Amsterdam, The Netherlands

Hypothesis

TG2 is involved in astrocyte-fibronectin interactions.

Conclusion

Astrocyte-derived TG2 contributes to the interaction between astrocytes and fibronectin, potentially regulating ECM remodeling and glial scarring.

Supporting Evidence

  • TG2 expression and activity increased after treatment with pro-inflammatory cytokines.
  • Active TG2 on the surface of astrocytes enhances adhesion to fibronectin.
  • Inhibition of TG2 activity reduces astrocyte adhesion and migration.
  • Astrocyte-derived TG2 interacts with fibronectin, contributing to ECM remodeling.
  • Cytokine treatment alters astrocyte migration capacity.

Takeaway

This study shows that a protein called TG2 helps brain cells called astrocytes stick to a special protein called fibronectin, which is important for healing in the brain.

Methodology

Primary rat astrocytes were treated with cytokines and analyzed for TG2 expression, adhesion, and migration on fibronectin.

Potential Biases

Potential bias in interpreting results due to the use of specific inhibitors and cytokine treatments.

Limitations

The study primarily uses rat astrocytes, which may not fully represent human astrocyte behavior.

Participant Demographics

Newborn Dark Agouti rats were used for astrocyte cultures.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0025037

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