Small ubiquitin-like modifier 4 (SUMO4) polymorphisms and Vogt-Koyanagi-Harada (VKH) syndrome in the Chinese Han population
2008

SUMO4 Polymorphisms and Vogt-Koyanagi-Harada Syndrome in Chinese Han Population

Sample size: 533 publication Evidence: moderate

Author Information

Author(s): Hou Shengping, Yang Peizeng, Du Liping, Zhou Hongyan, Lin Xiaomin, Liu Xiaoli, Kijlstra Aize

Primary Institution: The First Affiliated Hospital, Chongqing Medical University

Hypothesis

Are SUMO4 polymorphisms associated with Vogt-Koyanagi-Harada syndrome in the Chinese Han population?

Conclusion

HLA-DR4 and HLA-DRw53 are strongly associated with susceptibility to VKH syndrome, but SUMO4 polymorphisms are not.

Supporting Evidence

  • HLA-DR4 and HLA-DRw53 were significantly associated with susceptibility to VKH syndrome.
  • A decreased frequency of SUMO4 +438 TT genotype was found in VKH patients compared with healthy controls.
  • Stratification analysis did not show any associations between SUMO4 polymorphisms and VKH syndrome.

Takeaway

The study looked at whether certain gene variations are linked to a disease called VKH syndrome in Chinese people, but found that they are not.

Methodology

Genotyping for SUMO4 polymorphisms was performed on 231 VKH patients and 302 controls using PCR-RFLP.

Limitations

The significance of some findings was lost after Bonferroni correction, and the sample size for HLA-DRw53 negative patients was small.

Participant Demographics

231 VKH patients (128 males, 103 females) and 302 healthy controls (164 males, 138 females), all Chinese Han.

Statistical Information

P-Value

p=3.21×10−16

Confidence Interval

95% CI=6.99–26.98

Statistical Significance

p<0.05

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