Intrathecal lidocaine pretreatment attenuates immediate neuropathic pain by modulating Nav1.3 expression and decreasing spinal microglial activation
2011

Lidocaine Pretreatment Reduces Neuropathic Pain in Rats

Sample size: 66 publication 10 minutes Evidence: moderate

Author Information

Author(s): Cheng Kuang-I, Lai Chung-Sheng, Wang Fu-Yuan, Wang Hung-Chen, Chang Lin-Li, Ho Shung-Tai, Tsai Hung-Pei, Kwan Aij-Li

Primary Institution: Kaohsiung Medical University, Kaohsiung, Taiwan

Hypothesis

Does intrathecal lidocaine pretreatment attenuate neuropathic pain after nerve injury?

Conclusion

Intrathecal lidocaine pretreatment can alleviate acute neuropathic pain for up to three days by modulating sodium channel expression and reducing spinal microglial activation.

Supporting Evidence

  • Intrathecal lidocaine reduced mechanical allodynia and thermal hyperalgesia in rats.
  • Nav1.3 expression was significantly attenuated in the lidocaine pretreated group.
  • Microglial activation was decreased in the spinal cord after lidocaine pretreatment.

Takeaway

Giving lidocaine before surgery helps reduce pain in rats with nerve injuries. It works by calming down certain cells in the spine that cause pain.

Methodology

Sixty-six adult male Sprague-Dawley rats were divided into three groups: sham, ligated, and pretreated with intrathecal lidocaine, and assessed for pain responses and sodium channel expression.

Limitations

The effects of lidocaine pretreatment were limited to three days and may not translate directly to human patients.

Participant Demographics

Adult male Sprague-Dawley rats weighing 300-350 g.

Statistical Information

P-Value

p = 0.003

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1471-2377-11-71

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