Characterization of highly frequent epitope-specific CD45RA+/CCR7+/- T lymphocyte responses against p53-binding domains of the human polyomavirus BK large tumor antigen in HLA-A*0201+ BKV-seropositive donors
2006

Study of Immune Responses to BK Virus Large Tumor Antigen

Sample size: 7 publication Evidence: moderate

Author Information

Author(s): Maurizio Provenzano, Laura Bracci, Stephen Wyler, Tvrtko Hudolin, Giovanni Sais, Rainer Gosert, Paul Zajac, Giorgio Palu', Michael Heberer, Hans H Hirsch, Giulio C Spagnoli

Primary Institution: University Hospital Basel, Switzerland

Hypothesis

The study investigates CD8+ T immune responses to BK virus large tumor antigen portions involved in p53 binding in HLA-A*0201+ BKV LTag experienced individuals.

Conclusion

The study found that there are widespread cellular immune responses against epitopes within the BK virus large tumor antigen that may play a role in immunosurveillance against tumors associated with BK virus infection.

Supporting Evidence

  • All tested HLA-A*0201+ BKV LTag experienced individuals showed epitope-specific immune responses.
  • LTag579–587 was naturally processed and induced cytotoxic responses.
  • CD8+ T cells were detectable only in the CD45RA+ subset.

Takeaway

The body can recognize parts of a virus that might cause cancer, and this study looked at how well our immune system can respond to those parts.

Methodology

The study used peptide stimulation of CD8+ T cells from BKV seropositive donors and assessed immune responses through IFN-γ gene expression and cytotoxicity assays.

Potential Biases

Potential bias may arise from the selection of donors and the specific focus on HLA-A*0201+ individuals.

Limitations

The study was limited to a small sample size and focused only on HLA-A*0201+ individuals.

Participant Demographics

Participants were HLA-A*0201+ Caucasians with a mean age of 36.8 years.

Statistical Information

P-Value

0.0001

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1479-5876-4-47

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