A Crosslinking Analysis of GAP-43 Interactions with Other Proteins in Differentiated N1E-115 Cells
2008

GAP-43 Interactions with Other Proteins in Neurons

publication Evidence: moderate

Author Information

Author(s): Ollom Callise M., Denny John B.

Primary Institution: University of Texas Health Science Center at San Antonio

Hypothesis

GAP-43 binds to various proteins in growing neurons as part of its mechanism of action.

Conclusion

GAP-43 does not extensively interact with cytoskeletal proteins in differentiated N1E-115 cells, but it may interact with calmodulin.

Supporting Evidence

  • GAP-43 was not found in the 100,000 × g pellet fractions, indicating it does not sediment with actin.
  • Calmodulin co-immunoprecipitated with GAP-43 following crosslinker treatment.
  • The majority of GAP-43 in growing N1E-115 cells is not complexed to other proteins.

Takeaway

GAP-43 is a protein that helps neurons grow, but in this study, it didn't stick to the usual partners like actin; instead, it mostly hung out with calmodulin.

Methodology

Differentiated N1E-115 neuroblastoma cells were treated with a crosslinking reagent and analyzed by immunoprecipitation and two-dimensional gel electrophoresis.

Limitations

The study does not confirm the presence of GAP-43 in complexes with actin or other proteins, leaving open the possibility of untested interactions.

Digital Object Identifier (DOI)

10.3390/ijms9091753

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