A patient with glycogen storage disease type Ib presenting with acute myeloid leukemia (AML) bearing monosomy 7 and translocation t(3;8)(q26;q24) after 14 years of treatment with granulocyte colony-stimulating factor (G-CSF): A case report
2008

Case of a Patient with Glycogen Storage Disease Type Ib and Acute Myeloid Leukemia

Sample size: 1 publication Evidence: low

Author Information

Author(s): Thomas Schroeder, Barbara Hildebrandt, Ertan Mayatepek, Ulrich Germing, Rainer Haas

Primary Institution: Heinrich-Heine-University, Düsseldorf, Germany

Hypothesis

Does long-term treatment with granulocyte colony-stimulating factor increase the risk of acute myeloid leukemia in patients with glycogen storage disease type Ib?

Conclusion

Regular bone marrow examinations should be conducted for patients with glycogen storage disease type Ib on long-term G-CSF treatment due to the increased risk of developing acute myeloid leukemia.

Supporting Evidence

  • The patient had a history of glycogen storage disease type Ib and received G-CSF for 14 years.
  • Cytogenetic analysis revealed monosomy 7 and translocation t(3;8)(q26;q24).
  • The patient developed acute myeloid leukemia after long-term G-CSF treatment.

Takeaway

A patient with a rare disease developed leukemia after many years of treatment, showing that the treatment might have caused the cancer.

Methodology

Case report detailing the patient's medical history, treatment, and outcomes.

Potential Biases

Potential bias due to the retrospective nature of the case report.

Limitations

The findings are based on a single case, limiting generalizability.

Participant Demographics

28-year-old Turkish male.

Digital Object Identifier (DOI)

10.1186/1752-1947-2-319

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