Fisetin Lowers Methylglyoxal Dependent Protein Glycation and Limits the Complications of Diabetes
2011

Fisetin Reduces Diabetic Complications

Sample size: 24 publication 10 minutes Evidence: moderate

Author Information

Author(s): Pamela Maher, Richard Dargusch, Jennifer L. Ehren, Shinichi Okada, Kumar Sharma, David Schubert

Primary Institution: The Salk Institute for Biological Studies

Hypothesis

Can fisetin prevent the complications of diabetes in vivo by targeting multiple pathways associated with hyperglycemia?

Conclusion

Fisetin lowers the elevation of methylglyoxal-dependent protein glycation associated with diabetes and ameliorates multiple complications of the disease.

Supporting Evidence

  • Fisetin reduced kidney hypertrophy and albuminuria in diabetic mice.
  • Fisetin increased glyoxalase 1 enzyme activity.
  • Fisetin decreased markers of oxidative stress and inflammation in diabetic mice.
  • Fisetin improved locomotor activity in the open field test, indicating reduced anxiety.

Takeaway

Fisetin is a natural compound that helps reduce problems caused by diabetes in mice, like kidney damage and anxiety.

Methodology

Akita mice were fed a diet containing fisetin for 18 weeks, and various health parameters were measured.

Potential Biases

Potential bias in the selection of animal models and the interpretation of results.

Limitations

The study was conducted in a mouse model, which may not fully replicate human diabetes.

Participant Demographics

Male Akita mice, a model of type 1 diabetes.

Statistical Information

P-Value

p<0.001

Statistical Significance

p<0.001

Digital Object Identifier (DOI)

10.1371/journal.pone.0021226

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication