Genome-wide analysis of KAP1 binding suggests autoregulation of KRAB-ZNFs
2007

KAP1 Binding and Autoregulation of KRAB-ZNFs

Sample size: 26000 publication 10 minutes Evidence: high

Author Information

Author(s): O'Geen Henriette, Squazzo Sharon L, Iyengar Sushma, Blahnik Kim, Rinn John L, Chang Howard Y, Green Roland, Farnham Peggy J

Primary Institution: University of California Davis

Hypothesis

Does KAP1 mediate the trimethylation of histone H3 on lysine 9 and regulate the expression of KRAB-ZNF genes?

Conclusion

KAP1 is involved in the autoregulation of KRAB-ZNF gene expression through its binding to their transcribed regions.

Supporting Evidence

  • KAP1 binding sites were identified in the transcribed regions of ZNF genes.
  • H3me3K9 and H3me3K27 target different classes of transcription factors.
  • Many KAP1 target genes are also bound by H3me3K9.
  • ZNF genes are the most highly enriched class of KAP1 target genes.
  • KAP1 represses transcription of many KRAB-ZNF genes.

Takeaway

This study found that a protein called KAP1 helps control the activity of certain genes by sticking to them, especially those that are important for making other proteins.

Methodology

The study used chromatin immunoprecipitation (ChIP)-chip assays to analyze KAP1 binding across ~26,000 human promoters.

Statistical Information

P-Value

5E−27

Statistical Significance

p<0.0001

Digital Object Identifier (DOI)

10.1371/journal.pgen.0030089

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