HIV-1 gp120 Induces Expression of IL-6 through a Nuclear Factor-Kappa B-Dependent Mechanism: Suppression by gp120 Specific Small Interfering RNA
2011

HIV-1 gp120 Increases IL-6 in Astrocytes

publication 10 minutes Evidence: high

Author Information

Author(s): Shah Ankit, Verma Ashish S., Patel Kalpeshkumar H., Noel Richard, Rivera-Amill Vanessa, Silverstein Peter S., Chaudhary Suman, Bhat Hari K., Stamatatos Leonidas, Singh Dhirendra P., Buch Shilpa, Kumar Anil

Primary Institution: University of Missouri-Kansas City

Hypothesis

Is gp120 involved in IL-6 over-expression in astrocytes?

Conclusion

HIV-1 gp120 induces IL-6 expression in astrocytes through the NF-κB pathway.

Supporting Evidence

  • gp120 transfection increased IL-6 mRNA by 51.3±2.1 fold.
  • IL-6 protein levels were significantly higher in gp120-treated cells.
  • Inhibitors of NF-κB reduced IL-6 expression by over 50%.
  • gp120-specific siRNA effectively blocked IL-6 expression.

Takeaway

HIV-1 gp120 makes brain cells called astrocytes produce more of a chemical called IL-6, which can cause inflammation in the brain.

Methodology

The study used human astrocyte cell lines and primary human fetal astrocytes to assess IL-6 expression after gp120 treatment, employing transfection and various inhibitors.

Limitations

The study primarily used cell lines, which may not fully replicate in vivo conditions.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0021261

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