Nitric Oxide Synthase Inhibition Enhances the Antitumor Effect of Radiation in the Treatment of Squamous Carcinoma Xenografts
2011

Nitric Oxide Synthase Inhibition Enhances Tumor Control with Radiation

Sample size: 29 publication 10 minutes Evidence: high

Author Information

Author(s): Cardnell Robert J. G., Mikkelsen Ross B.

Primary Institution: Virginia Commonwealth University

Hypothesis

Does the nitric oxide synthase inhibitor, NG-nitro-L-arginine (L-NNA), enhance the effectiveness of ionizing radiation in controlling squamous carcinoma tumor growth?

Conclusion

The combination of L-NNA and radiation significantly delays tumor growth and enhances survival in mice with squamous carcinoma xenografts.

Supporting Evidence

  • L-NNA treatment stopped tumor growth for at least 10 days.
  • The combination of L-NNA and radiation caused 82% tumor cell killing.
  • Animals receiving both L-NNA and radiation survived significantly longer than those receiving either treatment alone.

Takeaway

This study found that a special medicine can help radiation therapy work better against certain tumors, making them shrink and helping mice live longer.

Methodology

Mice with squamous carcinoma xenografts were treated with L-NNA and exposed to ionizing radiation, followed by analysis of tumor growth and survival.

Limitations

The study primarily focuses on mouse models, which may not fully replicate human responses.

Participant Demographics

Athymic NCr-nu/nu male mice, 5-6 weeks old.

Statistical Information

P-Value

0.00007

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0020147

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