Heavily glycosylated, highly fit SIVMne variants continue to diversify and undergo selection after transmission to a new host and they elicit early antibody dependent cellular responses but delayed neutralizing antibody responses
2008

SIV Variants Evolve After Transmission to New Hosts

Sample size: 6 publication 10 minutes Evidence: moderate

Author Information

Author(s): Dawnnica Eastman, Anne Piantadosi, Xueling Wu, Donald N. Forthal, Gary Landucci, Jason T. Kimata, Julie Overbaugh

Primary Institution: Fred Hutchinson Cancer Research Center

Hypothesis

Do SIV variants with higher replication fitness continue to diversify after transmission to a new host?

Conclusion

SIV variants that had high replication fitness and escaped neutralizing antibodies in one host continued to evolve upon transmission to a new host.

Supporting Evidence

  • SIV variants from late-stage infections showed more envelope sequence evolution than those from early-stage infections.
  • Changes in glycosylation sites occurred before neutralizing antibody responses were detected.
  • The study found that late-stage SIV variants had higher replication fitness and continued to evolve after transmission.

Takeaway

When SIV viruses move from one monkey to another, they keep changing and adapting, even if they were already strong in the first monkey.

Methodology

The study analyzed envelope sequence variation of SIVMne variants in macaques infected at different stages of infection.

Potential Biases

Potential bias due to the small number of animals and the specific strains used.

Limitations

The study had a small sample size, which may limit the generalizability of the findings.

Participant Demographics

Macaques infected with different stages of SIVMne variants.

Statistical Information

P-Value

p = 0.07

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1743-422X-5-90

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication