The Role of IL-15 Deficiency in the Pathogenesis of Virus-Induced Asthma Exacerbations
2011

The Role of IL-15 Deficiency in Virus-Induced Asthma Exacerbations

Sample size: 25 publication 10 minutes Evidence: moderate

Author Information

Author(s): Laza-Stanca Vasile, Simon D. Message, Michael R. Edwards, Hayley L. Parker, Mihnea T. Zdrenghea, Tatiana Kebadze, Onn M. Kon, Patrick Mallia, Luminita A. Stanciu, Sebastian L. Johnston

Primary Institution: Imperial College London

Hypothesis

IL-15 production may be deficient in asthma and related to asthma exacerbation pathogenesis.

Conclusion

Deficient IL-15 production in asthma may be important in the pathogenesis of asthma exacerbations.

Supporting Evidence

  • Rhinovirus infections are the major cause of asthma exacerbations.
  • IL-15 levels in bronchoalveolar lavage fluid were decreased in asthmatics.
  • Deficient IL-15 production was inversely related to lower respiratory symptom severity during rhinovirus infection.

Takeaway

This study found that people with asthma don't produce enough of a protein called IL-15 when they get a cold virus, which might make their asthma worse.

Methodology

The study investigated IL-15 induction by rhinovirus in macrophages from asthmatic and control subjects, measuring levels in bronchoalveolar lavage fluid and correlating with symptom severity.

Potential Biases

Potential bias in participant selection as all subjects were non-smokers and may not represent the broader asthma population.

Limitations

The study had a small sample size and may not generalize to all asthma patients.

Participant Demographics

15 non-atopic healthy and 10 atopic mild asthmatic subjects, all rhinovirus-16 serum neutralizing antibody negative.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.ppat.1002114

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