Peripheral anti-inflammatory effects explain the ginsenosides paradox between poor brain distribution and anti-depression efficacy
2011

Ginsenosides and Their Role in Reducing Depression-like Behavior

Sample size: 10 publication 10 minutes Evidence: moderate

Author Information

Author(s): Kang An, Hao Haiping, Zheng Xiao, Liang Yan, Xie Yuan, Xie Tong, Dai Chen, Zhao Qijin, Wu Xiaolan, Xie Lin, Wang Guangji

Primary Institution: Key Laboratory of Drug Metabolism and Pharmacokinetics, State Key Laboratory of Natural Medicines, China Pharmaceutical University, Nanjing, Jiangsu, China

Hypothesis

The central therapeutic effects of ginseng total saponins (GTS) might derive from peripheral anti-inflammation activities.

Conclusion

The anti-depression efficacy of GTS may be largely attributable to its peripheral anti-inflammatory activity.

Supporting Evidence

  • GTS significantly reduced LPS-induced depression-like behavior in mice.
  • GTS treatment decreased production of proinflammatory cytokines in both LPS-challenged mice and RAW264.7 cells.
  • GTS pretreatment attenuated increases in IDO activity associated with LPS-induced neuroinflammation.

Takeaway

Ginseng can help people feel less sad by reducing inflammation in the body, even though it doesn't get into the brain very well.

Methodology

The study used LPS-induced depression-like behavior models in mice, assessing the effects of GTS through various behavioral tests and cytokine measurements.

Limitations

The study primarily focused on peripheral effects and did not explore potential central effects of ginsenosides.

Participant Demographics

Male CD-1 mice, 10-12 weeks old.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1742-2094-8-100

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