Forced mobilization accelerates pathogenesis: characterization of a preclinical surgical model of osteoarthritis
2007

Forced Mobilization and Osteoarthritis in Rats

Sample size: 96 publication 10 minutes Evidence: high

Author Information

Author(s): Appleton C, Thomas G, McErlain David D, Pitelka Vasek, Schwartz Neil, Bernier Suzanne M, Henry James L, Holdsworth David W, Beier Frank

Primary Institution: CIHR Group in Skeletal Development & Remodeling, Schulich School of Medicine & Dentistry, The University of Western Ontario

Hypothesis

Forced mobilization in a rat model of osteoarthritis will accelerate the progression of the disease.

Conclusion

Forced mobilization accelerates osteoarthritis damage in the destabilized joint.

Supporting Evidence

  • Cartilage degradation and subchondral changes were observed as early as 2 weeks after surgery.
  • Forced mobilization exercise accelerated OA progression compared to non-mobilized controls.
  • Histologic changes correlated well with micro-CT analysis of underlying bone.
  • Increased chondrocyte hypertrophy was noted during OA pathogenesis.
  • The model reflects properties of human OA and is suitable for longitudinal studies.

Takeaway

This study shows that making rats exercise after a knee surgery makes their arthritis get worse faster.

Methodology

Male Sprague-Dawley rats underwent joint destabilization surgery and were divided into groups for forced mobilization or control.

Potential Biases

Potential bias in the assessment of histological changes due to subjective scoring.

Limitations

The model may not fully replicate human osteoarthritis and the effects of forced mobilization may vary.

Participant Demographics

Male Sprague-Dawley rats, weight-matched, aged 8-12 weeks.

Statistical Information

P-Value

0.029

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/ar2120

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