Histopathologic characterization of the BTBR mouse model of autistic-like behavior reveals selective changes in neurodevelopmental proteins and adult hippocampal neurogenesis
2011

Study of Brain Changes in BTBR Mice Related to Autism

Sample size: 34 publication 10 minutes Evidence: moderate

Author Information

Author(s): Diane T. Stephenson, Sharon M. O'Neill, Sapna Narayan, Aadhya Tiwari, Elizabeth Arnold, Harry D. Samaroo, Du Fu, Robert H. Ring, Brian Campbell, Mathew Pletcher, Vidita A. Vaidya, Daniel Morton

Primary Institution: Pfizer Global Research and Development

Hypothesis

More complete histopathological characterization of the BTBR brain would reveal abnormal cellular and anatomic features that may correlate with the behavioral deficits and callosal abnormalities.

Conclusion

The study found selective alterations in glia, neurons, and synapses in BTBR forebrain, along with reduced neurogenesis in the adult hippocampus.

Supporting Evidence

  • BTBR mice showed reduced neurogenesis in the hippocampus.
  • Significant reductions in markers of neurogenesis were observed.
  • Alterations in glial and neuronal markers were noted in the forebrain.
  • The study suggests potential therapeutic strategies for autism spectrum disorders.

Takeaway

Researchers looked at the brains of special mice that show behaviors like autism and found some changes in brain cells that might help us understand autism better.

Methodology

The study used immunohistochemistry and quantitative image analysis on brain sections from BTBR and control mice to assess various neurodevelopmental markers.

Potential Biases

Potential bias may arise from the use of a single mouse model and the interpretation of results in the context of human autism.

Limitations

The study primarily focused on histopathological changes and did not explore all potential neurobiological factors involved in autism.

Participant Demographics

The study involved male BTBR and C57Bl/6J control mice aged 8 to 10 weeks.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/2040-2392-2-7

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