Legionella pneumophila infection induces programmed cell death, caspase activation, and release of high-mobility group box 1 protein in A549 alveolar epithelial cells: inhibition by methyl prednisolone
2008

How Legionella pneumophila Causes Cell Death in Lung Cells

publication Evidence: moderate

Author Information

Author(s): Furugen Makoto, Higa Futoshi, Hibiya Kenji, Teruya Hiromitsu, Akamine Morikazu, Haranaga Shusaku, Yara Satomi, Koide Michio, Tateyama Masao, Mori Naoki, Fujita Jiro

Primary Institution: University of the Ryukyus

Hypothesis

The study investigates the mechanism by which Legionella pneumophila induces apoptosis in A549 alveolar epithelial cells and the effect of methyl prednisolone on this process.

Conclusion

Infection of A549 alveolar epithelial cells with Legionella pneumophila caused programmed cell death, activation of various caspases, and release of HMGB1.

Supporting Evidence

  • The virulent strain of Legionella pneumophila induced DNA fragmentation and activation of caspases in A549 cells.
  • Methyl prednisolone reduced DNA fragmentation and caspase activation in infected cells.
  • The avirulent strain did not cause significant cell death or caspase activation.

Takeaway

When a certain bacteria infects lung cells, it can make them die in a specific way, and a medicine called methyl prednisolone can help reduce this cell death.

Methodology

The study used TUNEL staining to assess DNA fragmentation and colorimetric assays to measure caspase activity in A549 cells infected with virulent and avirulent strains of Legionella pneumophila.

Limitations

The study was conducted in vitro, and further in vivo studies are needed to confirm the findings.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1465-9921-9-39

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