Type I interferon-dependent gene MxA in perinatal HIV-infected patients under antiretroviral therapy as marker for therapy failure and blood plasmacytoid dendritic cells depletion
2008

MxA as a Marker for Therapy Failure in HIV-Infected Children

Sample size: 33 publication Evidence: moderate

Author Information

Author(s): Badolato Raffaele, Ghidini Claudia, Facchetti Fabio, Serana Federico, Sottini Alessandra, Chiarini Marco, Spinelli Elena, Lonardi Silvia, Plebani Alessandro, Caimi Luigi, Imberti Luisa

Primary Institution: Istituto di Medicina Molecolare 'Angelo Nocivelli', Department of Pediatrics, University of Brescia, Brescia, Italy

Hypothesis

The study aims to determine the role of interferon-alpha in controlling HIV infection in perinatally infected patients under antiretroviral therapy.

Conclusion

MxA mRNA levels may serve as a valuable marker to monitor the clinical response to therapy in HIV patients.

Supporting Evidence

  • HIV replication unresponsive to antiretroviral treatment may lead to increased MxA mRNA expression.
  • MxA mRNA levels correlated with HIV RNA copy numbers.
  • Patients with low CD4+ counts showed severe depletion of plasmacytoid dendritic cells.

Takeaway

This study looked at how a specific gene, MxA, can help doctors understand if HIV treatment is working for kids who got the virus at birth.

Methodology

The study used flow cytometry, real-time PCR, and ELISA to analyze plasmacytoid dendritic cells and measure interferon-alpha and MxA mRNA levels.

Limitations

The study did not assess pDC counts before treatment and relied on a small sample size.

Participant Demographics

33 perinatally HIV-infected patients aged 2 to 19 years, with a mix of male and female participants.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1479-5876-6-49

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication