Metal-Dependent Cell Death in Renal Fibrosis: Now and in the Future
2024

Metal-Dependent Cell Death in Renal Fibrosis

publication 10 minutes Evidence: moderate

Author Information

Author(s): Li Te, Yu Chen, Couvineau Alain

Primary Institution: Tongji Hospital, School of Medicine, Tongji University, Shanghai, China

Hypothesis

The study explores the relationship between metal-dependent cell death and renal fibrosis.

Conclusion

The review highlights the complex interplay between metal ions and cell death mechanisms in the context of renal fibrosis, suggesting potential therapeutic strategies.

Supporting Evidence

  • Liproxstatin-1 reduces renal fibrosis by inhibiting ferroptosis.
  • Astragalus mongholicus Bunge and Panax notoginseng formula improves renal fibrosis by inhibiting ferroptosis signaling.
  • Kaempferitrin inhibits NOX4-mediated ferroptosis in tubular cells.
  • Nobiletin has anti-fibrotic effects in renal fibrosis models.
  • Tectorigenin mitigates renal fibrosis by inhibiting Smad3-mediated ferroptosis.

Takeaway

This study talks about how metals like iron and copper can cause kidney damage and lead to diseases, and it suggests ways to treat these problems.

Methodology

The review synthesizes existing literature on metal-dependent cell death mechanisms and their implications for renal fibrosis.

Limitations

The study primarily focuses on ferroptosis and may overlook other metal-induced cell death pathways.

Digital Object Identifier (DOI)

10.3390/ijms252413279

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