Deletion of the Mitochondrial Flavoprotein Apoptosis Inducing Factor (AIF) Induces β-Cell Apoptosis and Impairs β-Cell Mass
2009

The Role of AIF in β-Cell Health

Sample size: 6 publication 10 minutes Evidence: moderate

Author Information

Author(s): Schulthess Fabienne T., Katz Sophie, Ardestani Amin, Kawahira Hiroshi, Georgia Senta, Bosco Domenico, Bhushan Anil, Maedler Kathrin

Primary Institution: Centre for Biomolecular Interactions, University of Bremen

Hypothesis

What role does AIF play in the regulation of β-cell survival, mass and insulin secretion?

Conclusion

AIF is essential for maintaining β-cell mass and for oxidative stress response.

Supporting Evidence

  • AIF depletion resulted in a 3-fold decrease in β-cell mass at 2 weeks of age.
  • Hq mutant mice showed an 8-fold increase in β-cell apoptosis.
  • AIF deficiency did not impair glucose-stimulated insulin secretion.
  • Human islets with AIF knockdown showed an 8.8-fold increase in β-cell apoptosis.

Takeaway

AIF helps keep insulin-producing cells healthy, and without it, these cells can die more easily.

Methodology

The study used Harlequin mutant mice and human islets to analyze the effects of AIF depletion on β-cell mass and apoptosis.

Limitations

The study primarily focused on a specific mouse model and may not fully represent human physiology.

Participant Demographics

Human islets from six different donors were used.

Statistical Information

P-Value

p<0.001

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0004394

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication