IgG Fc Receptors Provide an Alternative Infection Route for Murine Gamma-Herpesvirus-68 MHV-68 in Macrophages
2007

How Antibodies Affect Murine Gamma-Herpesvirus Infection in Macrophages

Sample size: 20 publication 10 minutes Evidence: moderate

Author Information

Author(s): Rosa Gustavo T. Gillet, Laurent Smith, Christopher M. de Lima, Brigitte D. Stevenson, Philip G. Stevenson

Primary Institution: University of Cambridge

Hypothesis

Can herpesvirus still infect Fc receptor+ cells even when exposed to neutralizing antibodies?

Conclusion

IgG Fc receptors limit the neutralization of gamma-herpesviruses such as MHV-68.

Supporting Evidence

  • Immune sera blocked MHV-68 infection of fibroblasts but enhanced infection of macrophages.
  • Viral glycoprotein-specific monoclonal antibodies also enhanced infection.
  • MHV-68 appeared to be predominantly latent in macrophages regardless of Fc receptor engagement.

Takeaway

This study found that antibodies meant to block a virus can actually help it infect certain immune cells instead.

Methodology

The researchers tested the effect of immune sera on MHV-68 infection in various cell types, including fibroblasts and macrophages.

Limitations

The study primarily focused on in vitro conditions, which may not fully replicate in vivo scenarios.

Participant Demographics

Mice used were BALB/c and C57BL/6J, aged 6-8 weeks.

Digital Object Identifier (DOI)

10.1371/journal.pone.0000560

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