Significant association of SREBP-2 genetic polymorphisms with avascular necrosis in the Korean population
2008

SREBP-2 Gene Polymorphisms and Avascular Necrosis in Koreans

Sample size: 716 publication 10 minutes Evidence: moderate

Author Information

Author(s): Kim Tae-Ho, Baek Jeong-In, Hong Jung Min, Choi Su-Jin, Lee Hye-Jin, Cho Hyun-Ju, Park Eui Kyun, Kim Un-Kyung, Kim Shin-Yoon

Primary Institution: Kyungpook National University Hospital

Hypothesis

SREBP-2 activity is responsible for the development of avascular necrosis and the variants of SREBP-2 gene are associated with the susceptibility to AVN.

Conclusion

The study found significant associations between certain SREBP-2 gene polymorphisms and the susceptibility to avascular necrosis in the Korean population.

Supporting Evidence

  • The minor allele frequency of rs2267439 showed a significant protective effect on AVN.
  • Genotype frequencies of rs2267439 were significantly different from controls.
  • Rs1052717 and rs2267443 polymorphisms were significantly associated with AVN risk.
  • Further analysis showed significant associations in idiopathic and alcohol-induced subgroups.

Takeaway

This study looked at how certain genes might affect the chances of getting a bone disease called avascular necrosis, finding some genes that help protect against it and others that increase risk.

Methodology

The study genotyped four SNPs in the SREBP-2 gene in 443 AVN patients and 273 control subjects using single-based extension genotyping.

Potential Biases

Potential biases may arise from the selection of control subjects and the reliance on self-reported medical histories.

Limitations

The study was limited to a specific population (Korean) and may not be generalizable to other ethnic groups.

Participant Demographics

443 AVN patients (366 men, 77 women; average age 49.7) and 273 control subjects (206 men, 67 women; average age 52.1).

Statistical Information

P-Value

0.01

Confidence Interval

0.604–0.935

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1471-2350-9-94

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