Whole blood transcriptional profiling in ankylosing spondylitis identifies novel candidate genes that might contribute to the inflammatory and tissue-destructive disease aspects
2011

Gene Profiling in Ankylosing Spondylitis

Sample size: 36 publication 10 minutes Evidence: moderate

Author Information

Author(s): Fernando M Pimentel-Santos, Dário Ligeiro, Mafalda Matos, Ana F Mourão, José Costa, Helena Santos, Anabela Barcelos, Fátima Godinho, Patricia Pinto, Margarida Cruz, João E Fonseca, Henrique Guedes-Pinto, Jaime C Branco, Matthew A Brown, Gethin P Thomas

Primary Institution: CEDOC, Faculdade de Ciências Médicas da Universidade Nova de Lisboa

Hypothesis

Can whole blood transcriptional profiling identify candidate genes associated with ankylosing spondylitis?

Conclusion

The study validated a gene expression signature for ankylosing spondylitis and identified strong candidate genes involved in inflammation and joint destruction.

Supporting Evidence

  • 239 probes corresponding to 221 genes were identified as significantly different between patients and controls.
  • Thirteen of the validated genes were downregulated 1.3- to 2-fold.
  • The study utilized a multicenter approach with ethnically homogeneous patients.

Takeaway

Researchers looked at blood samples from people with ankylosing spondylitis to find genes that might be causing the disease, and they found some important ones.

Methodology

The study used whole-genome microarray analysis on blood samples from 18 active AS patients and 18 matched controls, followed by validation in a larger cohort using qPCR.

Potential Biases

Potential biases may arise from differences in blood collection and processing across multiple centers.

Limitations

The study may be limited by the small sample size and the potential for misclassification of samples.

Participant Demographics

18 active AS patients and 18 matched controls, with a second cohort of 78 AS patients and 78 controls.

Statistical Information

P-Value

<0.0005

Confidence Interval

80%

Statistical Significance

p<0.0005

Digital Object Identifier (DOI)

10.1186/ar3309

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