Magnitude of Alloresponses to MHC Class I/II Expressing Human Cardiac Myocytes is Limited by their Intrinsic Ability to Process and Present Antigenic Peptides
2003

Cardiac Myocytes and Their Ability to Present Antigens

publication Evidence: moderate

Author Information

Author(s): J. Bruce Sundstrom, Kimberley C. Jollow, Veronique Braud, Francois Villinger, Andrew J. McMichael, E. Clinton Lawrence, Edwin W. Ades, Aftab A. Ansari

Primary Institution: Emory University School of Medicine

Hypothesis

The weak allogenicity of cardiac myocytes is related to their limited ability to process and present antigenic peptides.

Conclusion

Cardiac myocytes have a limited capacity to present antigens due to low expression of MHC genes and their products.

Supporting Evidence

  • W-1 cells can present the influenza A matrix 1 peptide to specific T cells after IFN-g pretreatment.
  • Constitutive expression of MHC class II genes is significantly lower in W-1 cells compared to professional APCs.
  • IFN-g pretreatment increases the expression of some MHC class I genes in W-1 cells.

Takeaway

Cardiac cells struggle to show off pieces of viruses to the immune system, which makes it hard for them to get noticed and attacked.

Methodology

The study involved examining the ability of a human cardiac myocyte cell line to process and present nominal antigens, comparing it with professional antigen-presenting cells.

Limitations

The study primarily focused on a single cell line and may not represent all cardiac myocytes.

Digital Object Identifier (DOI)

10.1080/10446670310001642410

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication