Mammary Gland Development as a Sensitive End Point after Acute Prenatal Exposure to an Atrazine Metabolite Mixture in Female Long-Evans Rats
2007

Effects of Atrazine Metabolites on Mammary Gland Development in Rats

Sample size: 12 publication 10 minutes Evidence: moderate

Author Information

Author(s): Enoch Rolondo R., Stanko Jason P., Greiner Sara N., Youngblood Geri L., Rayner Jennifer L., Fenton Suzanne E.

Primary Institution: North Carolina Central University

Hypothesis

Does a mixture of atrazine metabolites cause developmental effects in Long-Evans rats following exposure late in pregnancy?

Conclusion

Acute exposure to atrazine metabolites during late pregnancy causes persistent alterations in mammary gland development of female offspring.

Supporting Evidence

  • Maternal weight gain was significantly reduced during the dosing period after ATR-only exposure.
  • Female offspring prenatally exposed to 100 mg ATR/kg bw were not statistically different in body weight from control pups.
  • Delayed mammary gland development was observed as early as postnatal day 4 in all treatment groups.

Takeaway

When pregnant rats were given a mix of chemicals from a common herbicide, their baby girls had problems with breast development that lasted into adulthood.

Methodology

Pregnant Long-Evans rats were given a mixture of atrazine metabolites at various doses during gestation days 15-19, and mammary gland development was assessed.

Limitations

The study focused only on female Long-Evans rats, which may limit the generalizability of the findings to other species or genders.

Participant Demographics

Timed-pregnant Long-Evans rats, aged 9-15 weeks.

Statistical Information

P-Value

p<0.01

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1289/ehp.9612

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