Macrophage Tropism and Cytopathicity of HIV-1 Variants Isolated Sequentially from a Long-Term Survivor Infected with nef-Deleted Virus
2007

HIV-1 Variants and Their Effects on Macrophages

Sample size: 6 publication 10 minutes Evidence: moderate

Author Information

Author(s): Gorry Paul R, McPhee Dale A, Wesselingh Steven L, Churchill Melissa J

Primary Institution: Macfarlane Burnet Institute for Medical Research and Public Health

Hypothesis

Can nef-deleted HIV-1 strains enhance macrophage tropism and cytopathicity during disease progression?

Conclusion

Nef-deleted HIV-1 strains can evolve to increase their cytopathic potential without enhancing replication capacity.

Supporting Evidence

  • All nef-deleted viruses replicated to low levels in peripheral blood mononuclear cells and macrophages.
  • One nef-deleted virus caused high levels of syncytia in macrophages similar to control viruses.
  • Five nef-deleted viruses isolated prior to AIDS onset caused only minimal cytopathicity.

Takeaway

Some types of HIV can change over time to become more harmful to certain immune cells, even if they don't multiply faster.

Methodology

The study analyzed the replication capacity and cytopathicity of six sequentially isolated nef-deleted HIV-1 variants from a long-term survivor.

Limitations

The study is limited by the small sample size and the focus on a single subject.

Participant Demographics

The study involved a long-term survivor of HIV-1 infection, specifically subject D36 from the Sydney blood bank cohort.

Digital Object Identifier (DOI)

10.2174/1874285800701010001

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication